An example for NADP+ dependent dehydrogenase is Phosphogluconate dehydrogenase. Succinate dehydrogenase. Acyl-CoA dehydrogenase. Alkaline phosphatase O None of these
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- Discuss the metabolic control for HMG CoA reductase synthesis and activityConsidering the metabolic disorder "Ethanol Sensitivity". Which of the following therapeutic approaches could result in improvement for an individual with this disorder? (Select 2 answers) O Pharmacological competitive inhibition of Ethanol Dehydrogenase O Pharmacological alloseteric inhibition of cytosolic acetylaldehyde dehydrogenase O Pharmacological activation of cytosolic aldehyde dehydrogenase O Pharmacological uncompetitive inhibition of mitochondrial acetylaldehyde dehydrogenaseasap
- Name: Hendric Nduwuba DNP and Cellular Respiration Summary https://www.youtube.com/watch?v=eLtRan2qFx0 Your assignment today is to summarize in two paragraphs (8 sentences total) the effects of dinitrophenol (DNP) on the human body. Make sure you explain what DNP is, the stages of cellular respiration, and how DNP alters the metabolism of the person using it (side effects). The following vocabulary terms need to be used: cellular respiration, adenosine triphosphate, and dinitrophenol (DNP). Your objective is to describe the role of ATP in cellular respiration in order to explain how DNP affects the rate of metabolism. The first paragraph is an introduction to the summary and the second paragraph is an explanation of the main idea. You will be graded on this criteria: main idea, explanation of cellular respiration, explanation of DNP, and paragraph structure.int IMLeagues | Provi... Time Remaining: 00:18:21 A Hide Time Remaining A Arsenic is a water soluble element that is a poison because it functions as an allosteric inhibitor of the citric acid cycle enzymes pyruvate dehydrogenase and succinate dehydrogenase. One of the consequences of arsenic poisoning is lactic acidosis from the buildup of lactate in the body. Why does arsenic treatment result in a buildup of lactate? Legend Pyruvate Acetyl Hydrogen COA SH + NAD+ Pyruvate dehydrogenase Co,+NADH, H* Adenosine Carbon ATR triphosphate CoA Охygen Guanosine CGTP triphosphate Sulfur Acetyl-CoA COA SH Q Coenzyme Q CoA Coenzyme A HCO3 +CATP Water NADH Nicotinamide adenine dinucleotide Citrate Pyruvate carboxylase Pyruvate dehydrogenase Enzyme ADP+Pi Citrate synthase Aconitase Oxaloacetate Water cis-Aconitate Water NADH, H* Aconitase Malate dehydrogenase NAD. D-Isocitrate NAD* Malate NADH, H* Isocitrate dehydrogenase CO2 Citric acid cycle Fumarase MacBook PrConsider the complete oxidation of the fatty acid 15:14 1. How many cycles of B-oxidation are required to catabolize the fatty acid 15:14 ? 2. How many molecules of acetyl-CoA are produced during B-cxidation from the fatty acid 15:149? 3. In total, how many molecules of NADH are produced via the citric acid cycle from fatty acid15:14 4. How many molecules of FADH2 are produced during B-oxidation of the fatty acid15:149 5. In total, how many molecules of FADH2 are produced from the complete oxidation of the fatty acid 15:1A? 6. How many molecules of ATP are produced from the complete re-oxidation of all NADH produced from the fatty acid15:14 7. How many net molecules of ATP are produced from the fatty acid15:14 8. In total, how many water molecules are produced during the oxidative phosphorylation process when starting with the fatty acid15:14
- Arrange the steps of breakdown of oleic acid (pictured below) in the correct order. 1 2 3 4 5 activation by acyl-CoAsynthe ✓ [Choose ] one cycle ofß-oxidationbeginning at the enoyl-CoAhydratasestep three cycles ofẞ-oxidation five cycles ofß-oxidation activation by acyl-CoAsynthetase enoyl-CoAisomeraseactivity enoyl-CoAisomeraseactivity V one cycle ofß-oxidationbegini V five cycles ofß-oxidation OHPage > of 6 ZOOM + TCA cycle, oxidative phosphorylation, metabolic regulation TCA cycle (Ch. 16) 1. The citric acid cycle has eight enzymes: citrate synthase, aconitase, isocitrate dehydrogenase, a-ketoglutarate dehydrogenase, succinyl-CoA synthetase, succinate dehydrogenase, fumarase, and malate dehydrogenase. (a) Write a balanced equation for the reaction catalyzed by each enzyme. (b) Name the cofactor(s) required by each enzyme reaction. (c) For each enzyme determine which of the following describes the type of reaction(s) catalyzed: condensation (carbon–carbon bond formation); dehydration (loss of water); hydration (addition of water); decarboxylation (loss of CO2); oxidation-reduction; substrate-level phosphorylation; isomerization. (d) Write a balanced net equation for the catabolism of acetyl-CoA to CO2.Consider the complete oxidation of the fatty acid 17:1A9 1. How many cycles of B-oxidation are required to catabolize the fatty acid 17:1A? 2. How many molecules of acetyl-CoA are produced during B-oxidation from the fatty acid 17:1A ? 3. In total, how many molecules of NADH are produced via the citric acid cycle from fatty acid 17:1A ? 4. How many molecules of FADH2 are produced during B-oxidation of the fatty acid 17:1A 5. In total, how many molecules of FADH2 are produced from the complete oxidation of the fatty acid 17:1A ? 6. How many molecules of ATP are produced from the complete re-oxidation of all NADH produced from the fatty acid 17:149 7. How many net molecules of ATP are produced from the fatty acid 17:1A9 8. In total, how many water molecules are produced during the oxidative phosphorylation process when starting with the fatty acid 17:1A9
- Of the allosteric enzymes listed below, which are suppressed by NADH? Select all that apply. the pyruvate dehydrogenase complex a-ketoglutarate dehydrogenase complex U isocitrate dehydrogenase pyruvate kinase Ophosphofructokinase hexokinase Oglucokinasea) describe the overall reaction catalyzed by the pyruvate dehydrogenase Situational Problems I. Oxidative decarboxylation of pyruvate and the TCA cycle in muscles are stimulated by increased aerobic excrcise. These processes operate only when O, is present, although oxygen does not participate directly in these processes. Explain why oxidative decarboxylation of pyruvate is activated under aerobic conditions. For the answer: a) describe the overall reaction catalyzed by the pyruvate complex (PDH) and its regulation; b) outline the intermediates and enzymes of the TCA cycle; c) explain the relationship between the reactions of PDH and the TCA cycle and the respiratory chain.19) Consider the table of allosteric effectors and their effect on the following metabolic processes. Glycolysis Gluconeogenesis PDH complex Krebs cycle acetyl-CoA Not applicable AMP Not applicable + glucagon Not applicable Not applicable - Each row contains one error. Correct the error in the table below Table 2 Correction (indicate process and effect (+ or -) acetyl-CoA AMP glucagon - Briefly explain the effect of each allosteric effector on the corrected pathway (based on Table 2 above)