You are working on a project in which you want to transform exocrine cells into endocrine cells without passing the cells to pluripotent stage. What cell replacement therapy technique will you use
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You are working on a project in which you want to transform exocrine cells into endocrine cells without passing the cells to pluripotent stage. What cell replacement therapy technique will you use
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- Which is true for cancer cells: 1) Cell death occurs after a determined number of cell divisions 2) Contact with other cells reduces chance of cell division 3) Cell division occurs in the presence of stop signals.Put the following types of stem cells in order from MOST useful in regenerative medicine to LEAST useful. Group of answer choices adult--multipotent--pluripotent--totipotent totipotent--pluripotent--multipotent--adult adult--pluripotent--multipotent--totipotent adult--totipotent--multipotent--pluripotent pluripotent--multipotent--totipotent--adultIf a drug interferes with the formation of microtubules , its effectiveness must be related to disruption of mitotic spindle formation suppression of cyclin production inhibition of DNA synthesis myosin denaturation and inhibition of cleavage formation
- Neuropathy is often a side effect of anti-cancer drug treatments. Which of the following drugs induce neuropathy by disrupting microtubule formation thereby inhibiting axonal transport? Select ALL that apply Paclitaxel Oxaliplatin Vincristine Bortezomib Thalidomide CisplatinStem cells have the potential to transform into any of the various cell types in the body, and thus researchers have explored how stem cells may be used to treat different conditions. Which conditions benefit most from this treatment? At least 351 companies in the United States are now offering unapproved stem cell procedures at 570 clinics. If you had a serious illness such as Parkinson's disease and found an unregulated clinic that offered to cure your illness using stem cell therapy, would you accept the offer? What do you think is the reason that FDA and state medical boards are indifferent to those treatment; are unaware of the scope of the problem or simply are ignoring itYou are in charge of a new gene therapy clinic. Two cases have been referred to you for review and possible therapy. Case 1. A mutation in the promoter of a proto-oncogene causes the gene to make too much of its normal product, a receptor protein that promotes cell division. The uncontrolled cell division has caused cancer. Case 2. A mutation in an exon of a tumor-suppressor gene makes this gene nonfunctional. The product of this gene normally suppresses cell division. The mutant gene cannot suppress cell division, and this has led to cancer. What treatment options can you suggest for each case?
- You are working in a cell biology lab that investigates non-small cell lung cancer cells, which of these cellular features will be suggestive of senescence in the cells observed? Choose all that apply: Group of answer choices Large flattened morphology Reduced incorporation of 5-bromodeoxyuridine (in DNA replication) Increased p53 expression Decreased expression of p15INK4BThe best strategy for treating a specific type of human tumor can depend on identifying the type of cell that became cancerous to give rise to the tumor. For some tumors that have colonized a distant location (metastasized), identifying the parental cell type can be difficult. Because the type of IF protein expressed is cell-type-specific, using monoclonal antibodies that react with only one type of IF protein can help in this identification. What IF proteins would you produce monoclonal antibodies against to identify (a) a sarcoma of muscle cell origin, (b) an epithelial cell carcinoma, and (c) an astrocytoma (glial cell tumor)?What is the most likely outcome is we lose the tumor suppressor proteins, cyclin- dependent kinase inhibitors. Select one: o a. Cyclin-cylin dependent kinases will phosphorylate retinoblastoma protein and cell- cycle will not proceed. o b. Cyclin-cylin dependent kinase complex will not phosphorylate retinoblastoma protein and cell-cycle will not proceed. o c. Cyclin-cylin dependent kinase complex will not phosphorylate retinoblastoma protein and cell-cycle will proceed. o d. Cyclin-cylin dependent kinases will phosphorylate retinoblastoma protein and cell- cycle will proceed.
- My question is two fold. What are some diseases that cause and/or are caused by apoptosis? Also if apoptosis is the most common form of programmed cell death in animals, what other forms of programmed cell death are there? Thank you.What does cancer drugs play in interrupting mitosis of cancer cells? How do these drugs do that?If you were to compare stem cells to an everyday object or situation, what would they be like and why? I need some examples for this. It's very hard to think of one.