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Why doesn’t an action potential that is propagated down an axon go in the reverse direction ( give a molecular explanation; what channels are involved)
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- A common membrane-bound intermediary between the receptor and the effector protein within the plasma membrane is the __________________.Name the three phases of an action potential. Describe for each the underlying molecular basis and the ion involved. Why is the term voltage-gated channel applied to Na+ channels involved in the generation of an action potential?In the figure to the left, name the 4 phases of the action potential (Note: you have to write in where phase 4 occurs). Describe what happens in each phase with a focus on Na+ and K+ flow through channels and the membrane potential. Discuss the importance of threshold. How does this relate to the concept of APs being all or none?
- Draw details of the repolarization phase of an action potential from the following descriptions of the sequences of AfterHyperPolarization (AHP) and AfterDePolarization (ADP) sequences. Make the distinct phases clear and noticeable (5 % each) A complex AHP consisting of a first component AHP, an ADP, and a second component AHP before repolarization to resting membrane potential a first fast AHP component, followed by a slower AHP, followed by a fast ADP, and a second late AHP component before repolarization to restYou generate action potentials in a neuron bathed in solution in a petri dish by applying a threshold-level depolarizing stimulus near its axon hillock. If the solution surrounding the neuron contained 5mM K+, 150 mM Na+, and 0 mM Ca2+ which of the following would you expect? The neuron would not be able to propagate action potentials down the entire length of the axon The neuron would not release neurotransmitter from the axon terminal The neurons action potentials would have an unusually long duration The neuron would have a resting membrane potential of zeroIf a neuron is stimulated simultaneously by 2 stimuli at its dendrites (one generates a very large hyperpolarizing potential and the other generates a very small depolarizing potential). What would happen to this neuron? Group of answer choices The summation of these two graded potentials will generate an action potential and this action potential will travel down the axon. Summation of these two graded potentials will cause the membrane potential at the axon hillock to be more negative. These graded potentials will bring the resting membrane potential to 0 mV. These graded potentials with opposite polarity would therefore generate two action potentials and they will travel along this neuron in opposite direction.
- Assume presynaptic excitatory neuron A terminates on a postsynaptic cell near the axon hillock and presynaptic excitatory neuron B terminates on the same postsynaptic cell on a dendrite located on the side of the cell body opposite the axon hillock. Explain why rapid firing of presynaptic neuron A could bring the postsynaptic neuron to threshold through temporal summation, thus initiating an action potential, whereas firing of presynaptic neuron B at the same frequency and the same magnitude of EPSPs may not bring the postsynaptic neuron to threshold. thanks a lot in advance:)Local anesthetic is often used in dentistry to lessen the sensation of pain during dental procedures. It prevents propagation of nerve conduction by decreasing the flow of ions. a. Describe how depolarization of an action potential can be blocked by the use of local anesthetic. b. Your classmate explained that once the threshold potential has been reached, the inside charge of the neuron is more positive than the outside charge. Is she correct? Can you compare the inside charge to the outside charge in an axon treated with local anesthetic?Why does an action potential move in an all-or-nothing fashion down the length of an axon without stopping?
- Because the long axons of neurons look like electrical wires, and both neurons and electrical wires conduct electricity, it is tempting to equate the two. Compare and contrast the functioning of axons and electrical wires in terms of their structure and the nature of the electrical signals they conduct. Please include references.Depression is a condition in which sufferers may feel low mood, lack of motivation and sleep. It is caused by a lack of serotonin in the synaptic cleft of neurone pathways. Serotonin is a neurotransmitter. a) Describe how serotonin acts as a neurotransmitter. Using your understanding of synaptic transmission, explain how communication between neurones in serotonin pathways of the brain would be affected in someone with depression. b) Doctors use tubocurarine drug as an anaesthetic as it temporarily paralyses muscles. It blocks receptors at neuromuscular junctions. Why does this lead to paralysis?Let’s say the synapses of a neuron’s dendrites are filled with glutamate-gated channels which, when activated by glutamate, cause an excitatory postsynaptic potential (EPSP) in the neuron. In one instance, glutamate is released at all of the synapses simultaneously and this leads to an action potential in the neuron. A) What type of summation of PSPs is this? Why? B) List the sequence of steps that occur starting from binding of glutamate to the glutamate-gated channels at the synapses and ending with the membrane potential at the axon hillock returning to resting potential at the end of the action potential. (Include all the changes in voltage-gated channels underlying the action potential.)