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Well, the centriole is not present in the Pro-cariotic cell, nor is it in the permanent cell. So even if Amitosis can occur in procariotic cells, why not Amitosis occur in permanent cells? What is the thing that does not allow the permanent cell to divide normally in Amitosis like procariotic cell?
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- Why do you think apoptosis occurs by a different mechanism from the cell death that occurs in necrosis? What might be the consequences if apoptosis were not achieved in such a neat and orderly fashion, whereby the cell destroys itself fron within and avoids leakage of its contents into the extracellular space?Cytochalasin D inhibits the formation of microfilaments. Which of the following biological activities will be affected? Select all that apply. Formation of nuclear lamin network Formation of cleavage furrow in telophase U Cytosolic transport of secretory vesicles Movement of phagocytic cellsWhich of the following is not true about phagocytosis?(a) It is a form of exocytosis.(b) It is energy dependent.(c) It only occurs in eukaryotes.(d) A larger cell engulfs a smaller cell that will eventually bepresent in an internal vacuole.(e) It requires fusion of internal lysosomes to engulfed vac-uole for contents to be digested
- Which of the following best describes a way in which a normal growth factor can stimulate cell division? A A growth factor can bind to membrane receptors and initiate a signal transduction path- way that causes the synthesis of division-facilitating enzymes. B с D A growth factor can supply necessary biomolecular building blocks for cell division by diffusing through the cell membane. A growth factor can facilitate the transport of glucose into the cell, thereby providing it with nutrients to support cell division. A growth factor can inhibit specific checkpoints in the cell cycle and allow the cell to skip or bypass certain cell cycle phases.List and describe the three main types of cytoskeleton. If you wanted to do immunocytochemistry to specifically stain each type of cytoskeleton, what is a protein that could be used for each cytoskeletal type (in other words, what is a unique protein for each cytoskeletal type)? What are three types of actin structures? Describe the involvement of actin structures in cell migration. How is the growth and shrinking of microtubules regulated? Then describe the roles of microtubules in: chromosomal separation during mitosis and the movement of organelles and vesicles within a cell. Describe a possible mechanism on how signaling pathways might impact the cytoskeleton, so that cell migration could be regulated in a localized manner within a multicellular organism. (hint: think about the possible transcriptional regulation of the G-protein regulators) What are 2 main challenges of protein targeting? Then describe one way these challenges are overcome during signal-based targeting and one way…Secretory vesicles fuse with the cell membrane to release their contents to the outside of the cell. In this process, the membrane of the secretory vesicle becomes part of the cell membrane. Because small pieces of membrane are continually added to the cell membrane, we would expect the cell membrane to become larger and larger as secretion continues. However, the cell membrane stays the same size. Explain how this happens.
- Some drugs can prevent the formation of new lysosomes. However, a cell would continue to make hydrolytic enzymes from the DNA still contained in the cell. Where would you most likely find these enzymes after the drug treatment? O Golgi Complex O Smooth Endoplasmic Reticulum O Nucleus O Mitochondrion Strikingly different signaling systems can operate in similar ways. For example, ethylene gas released by ripening fruits diffuses through the air from plant toSome scientists claim that prokaryotes like bacteria have membrane bound organelles, like chromatophores, anammoxosomes and magnetosomes. What really is the truth because it is widely known that prokaryotes do not have membrane bound organelles. Are these claims true or merely speculative? If this is true, what is the physiological basis for this?Fibroblasts are motile cells that “creep” about in the connective tissue of the body where they play a number of roles, including secretion of the extracellular matrix and initiating certain wound healing events. These cells do not usually have microvilli nor do they express the actin-binding protein villin; both of these properties are characteristic of certain epithelial cells, such as those lining the intestine and kidneys. Remarkably enough, however, if one artificially engineers fibroblast cells to produce the villin protein, microvilli form on the fibroblasts. Given what you know about the function of villin, why might this striking change in cell morphology occur?
- Certain cells that line the stomach synthesize a digestive enzyme and then release it in large amounts into the stomach. Which of the following processes could be responsible for its release out of the cell? O phagocytosis O endocytosis O exocytosis O pinocytosisMicrovilli are commonly seen in cells involved in absorption of materials (like intestinal cells) because: none of the above they move cells forward they produce greater surface area of plasma membrane they move substances forwardWhy is cell furrowing important in cell division? If cytokinesis did not occur, what would be the end result?