Somo mutations, of changes in the sequenco of DNA, do not have any offect on the characteristios of the organiam. Why is this O The coll recognizes mutations and ignores therm when expressing the gene. O The immune syatom ropairs the mutated sequence during dovelopment. O The mutatod sequence still codes for the samo amino acid O The protein built from this mutated sequonico io deactivated by the cell.

Somo mutations, of changes in the sequenco of DNA, do not have any offect on the characteristios of the organiam. Why is this O The coll recognizes mutations and ignores therm when expressing the gene. O The immune syatom ropairs the mutated sequence during dovelopment. O The mutatod sequence still codes for the samo amino acid O The protein built from this mutated sequonico io deactivated by the cell.

Biology: The Unity and Diversity of Life (MindTap Course List)

15th Edition

ISBN:9781337408332

Author:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Publisher:Cecie Starr, Ralph Taggart, Christine Evers, Lisa Starr

Chapter10: Control Of Gene Expression

Section: Chapter Questions

Problem 4CT: The photos below show flowers from two Arabidopsis plants. The plant on the left is wild-type...

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ocument/d/1J-wo90GpYsd_jQSUBtDQHWisqGvSOteUQYoXaXazyS0/edit uction to cell.. R 1 Summary of Philo... E Petrona Andres Mig.. 2 Translations IXL: Par... IXL - Translations: g.. 1 IXL- meostasis Lab Exercise Tools Add-ons Help Last edit was 2 days ago text Calibri 12 BIU Conclusion: 1. List the changes you observed in the body color and perspiration level in response to? 2. Explain how the changes help the body adjust to maintain equilibrium (homeostasis)? 3. Speculate why a change in body temperature occurs? 4. Name which mechanisms your body uses to maintain a constant body temperature? 5. Explain why an increased breathing rate accompanies exercise? 6. Explain why an increased heart rate accompanies exercise? 7. Write a paragraph about the conclusions you can draw about your body's ability to maintain equilibrium (homeostasis). Be sure to include the answers to the questions above.
DNA Domiye Gorgous Shing myskery Jevel Lovely' Stop Cell yode and repuir DNA when it is time for the cell to divide, myseny prokin heps Jewel let time for tell divistn uf Lovely so that lokely can imore the Cell into S phace. Howeler, f DNA Damae is detetedl, Gosgeou antin goes on high akrt and actimles Shiny, Shiay then neutalizes Mysker, so thut Jerel remans attahed 2to haly uond loidy Camot aeibae lell cycle. Questons Describe hawa LOF mutaton in the shiny Dane wadd affecd this pathuay and the mpact it wadd have on cell cycle progression- 2) Dexribe haw a GOF mutation in the shing n Lodd affect this the imput it would have on cell cycle pogression would affect this pathway and 3) Basel on this, waad shiny be considered a tumor Suuprecer or a proto-onvogene.
Transfer of a donor chromosome fragment by a bacteriophage is defined as O Transduction O Budding O Conjugation O Transformation
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Queston you have discorered a gene called "leafh," whese proteia product leoks like a fruit. You sdlak sereral verbsions of the Gene: a wild type and two mutants. Motaton 1 causes the leafy protein to not work. Mutation2 prachuce lots of extr eafy protein. Which of these represents a GOF, ae' the wild type allele. LOF, 囚 Heatygene protein Teafa geset extra leaty pretein mutaticn 3 broken reafy aca muetafin o Qene protech Colum A LOF GOF wiled type Column B a.B b. C C. A 2. 3.
Which of the following would NOT be the right match? O Sickle cell anemia, a point mutation O nonsense mutation, lead to a nonfunctional protein O silent mutation, UCU into UCG serine O frameshift, insertion or deletion of chromosomes O missense mutation, does not effect the protein that much
Give me nucleotide sequence with pairing. Like this ATC TCA TGA GCC TAG AGT ACT. CGG
The photos below show flowers from two Arabidopsis plants. The plant on the left is wild-type (unmutated); the other carries a mutation that causes its flowers to have sepals and petals instead of stamens and carpels. The mutation inactivated one of the plant's ABC floral identity genes. Refer to Figure 10.8 and decide which gene (A, B, or C) has been inactivated.
Antibiotics and Protein Synthesis Antibiotics are molecules produced by microorganisms as defense mechanisms. The most effective antibiotics work by interfering with essential biochemical or reproductive processes. Many antibiotics block or disrupt one or more stages in protein synthesis. Some of these are mentioned here. Tetracyclines are a family of chemically related compounds used to treat several types of bacterial infections. Tetracyclines interfere with the initiation of translation. The tetracycline molecule attaches to the small ribosomal subunit and prevents binding of the tRNA anticodon during initiation. Both eukaryotic and prokaryotic ribosomes are sensitive to the action of tetracycline, but this antibiotic cannot pass through the plasma membrane of eukaryotic cells. Because tetracycline can enter bacterial cells to inhibit protein synthesis, it will stop bacterial growth, helping the immune system fight the infection. Streptomycin is used in hospitals to treat serious bacterial infections. It binds to the small ribosomal subunit but does not prevent initiation or elongation; however, it does affect the efficiency of protein synthesis. Binding of streptomycin changes the way mRNA codons interact with the tRNA. As a result, incorrect amino acids are incorporated into the growing polypeptide chain, producing nonfunctional proteins. In addition, streptomycin causes the ribosome to randomly fall off the mRNA, preventing the synthesis of complete proteins. Puromycin is not used clinically but has played an important role in studying the mechanism of protein synthesis in the research laboratory. The puromycin molecule is the same size and shape as a tRNA/amino acid complex. When puromycin enters the ribosome, it can be incorporated into a growing polypeptide chain, stopping further synthesis because no peptide bond can be formed between puromycin and an amino acid, causing the shortened polypeptide to fall off the ribosome. Chloramphenicol was one of the first broadspectrum antibiotics introduced. Eukaryotic cells are resistant to its actions, and it was widely used to treat bacterial infections. However, its use is limited to external applications and serious infections. Chloramphenicol destroys cells in the bone marrow, the source of all blood cells. In bacteria, this antibiotic binds to the large ribosomal subunit and inhibits the formation of peptide bonds. Another antibiotic, erythromycin, also binds to the large ribosomal subunit and inhibits the movement of ribosomes along the mRNA. Almost every step of protein synthesis can be inhibited by one antibiotic or another. Work on designing new synthetic antibiotics to fight infections is based on our knowledge of how the nucleotide sequence of mRNA is converted into the amino acid sequence of a protein. Questions Why are antibiotics ineffective in treating the common cold and other virus infections?
What would happen if a chemical interfered with the ability of RNA polymerase to bind to the DNA molecule? Multiple proteins would be created O The RNA Polymerase would bind to a different place on the DNA O Transcription could not take place O The process of making proteins would start with translation
oslorGulolaculGACu When does DNA replication occur in the cell cycle? Replicate this strand of DNA: ATTCGC TAG GU Aanine Transcribe the same piece of DNA Stop GU Valine Cystelne Stop Use the chart to create the amino acid sequence. Arginine AC Leucine Sorine C UG Lysine Proline Define gene mutation Asparagine True or False? All mutations are harmful. Define chromosomal mutation. Histidine Threonine
Proto-oncogenes can be converted to oncogenes in a numberof different ways. In some cases, the proto-oncogene itselfbecomes amplified up to hundreds of times in a cancer cell.An example is the cyclin D1 gene, which is amplified in somecancers. In other cases, the proto-oncogene may be mutatedin a limited number of specific ways, leading to alterations inthe gene product’s structure. The ras gene is an example of aproto-oncogene that becomes oncogenic after suffering pointmutations in specific regions of the gene. Explain why thesetwo proto-oncogenes (cyclin D1 and ras) undergo such differentalterations to convert them into oncogenes