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- Upon learning about the mechanism of apoptosis, the question asked was; Is cytochrome c being released from mitochondria or is it being expressed by an induced nuclear gene and being produced in the cytosol. Which one of the following observations would provide the best evidence that it was being released from the mitochondria? Bcl2 blocks release of cytochrome c from mitochondria and also inhibits apoptosis Active Fas receptors trigger release of cytochrome c from mitochondria. Extracellular signals that block apoptosis do not produce cytosolic cytochrome c Caspase inhibitors block release of cytochrome c from the mitochondriaArtificially induced apoptosis (controlled cellular death) is found to be an effective treat- ment for some forms of cancer. Which of the following describes the most likely mechanism by which apoptosis might be induced? A B с D by causing ribosomes to increase the rate of protein synthesis by triggering the division of mitochondria in the cell to increase ATP production by increasing the expression of membrane-bound glucose protein channels by causing lysosomes to release digestive enzymes into the cytosolHow do mitochondria initiate apoptosis?
- describe the role of mitochondria in initiating apoptosis. please describe and summarize in short two paragraphs only.p53 can trigger apoptosis if certain cell-cycle events fail. How does this regulatory outcome benefit a multicellular organism?Overexpression of the Myc protein is a common feature of many types of cancer cells, contributing to their excessive cell growth and proliferation. By contrast, when Myc is overexpressed in most normal cells, the result is not excessive proliferation, but cell-cycle arrest or apoptosis.Which one of the following statements provides the most likely explanation for why overexpression of Myc can have such different outcomes in normal cells and in cancer cells? A. Normal cells contain checks and balances that prevent Myc-induced proliferation. B. In normal cells, Myc protein acts as a mediator in cell-cycle arrest and apoptosis. C. The target protein for Myc-induced proliferation is missing from most normal cells. D. In normal cells, when Myc is overexpressed, the excess Myc protein precipitates.