Describe in your own words how nuclear transport and G protein coupled receptor (GPCR) signaling will be affected if GTP levels suddenly fall in body cells. Explain the importance of GTP in nuclear import/export and vesicular formation. Please answer asap and type your answer and do not copy from anywhere please
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- (Choose the best answer to complete the following sentence.) Transphosphorylation of receptor tyrosine kinases: activates JAK2 inhibits catalytic activity generates binding sites for signaling molecules triggers release of ligandThe synaptosome-associated receptors referred to as v-SNARES are transmembrane proteins located on which of the following eukaryotic cell structures? mitochondrial membranes chloroplast membranes vesicle membranes lysosomal membranes peroxisomal membranesA temperature-sensitive mutant yeast strain stops dividing when shifted from 25°C to 37°C. These cells are analyzed at different temperatures by a machine that measures the amount of DNA they contain, and the following graphs are obtained. number of cells number of cells [ Select] [Select] [Select] 25°C [Select] مل Cells in G1 phase should have [Select] Which of the following would explain the behavior of your mutant? Mark yes or no for each of the following choices. 2 amount of DNA/cell (arbitrary units) 37°C 1 2 amount of DNA/cell (arbitrary units) unit(s) amount of DNA. ✓ Inability to initiate DNA replication Defect in chromosome condensation ✓Defect in centrosome duplication ✓Defect in cytokinesis
- When epidermal growth factor (EGF)—the ligand for the epidermal growth factor receptor (EGFR)—binds EGFR on the cell surface, the intracellular domain of the receptor changes shape into an "active state.” The receptor then induces a cascade of kinases in the cytoplasm, resulting in the activation of mitogen-activated protein kinase (MAPK). One effect of EGFR signaling is the stimulation of cell proliferation. Overactive EGFR is frequently found in cancer cells. One strategy for developing anticancer drugs is to target the overactive signaling proteins found in cancer cells. Antibodies are proteins made by immune cells that only recognize specific biomolecules, called antigens. The major component of an anticancer drug is an antibody that specifically recognizes and binds EGFR. Which of the following will most likely happen when this anticancer drug is applied to a tumor that has a high level of EGFR? Tumor cells with active EGFR will increase and MAPK activity in the tumor will…Figure 9.8 HER2 is a receptor tyrosine kinase. In 30 percent of human breast cancers, HER2 is permanently activated, resulting in unregulated cell division. Lapatinib, a drug used to treat breast cancer, inhibits HER2 receptor tyrosine kinase autophosphorylation (the process by which the receptor adds phosphates onto itself), thus reducing tumor growth by 50 percent. Besides autophosphorylation, which of the following steps would be inhibited by Lapatinib? Signaling molecule binding, dimerization, and the downstream cellular response. Dimerization, and the downstream cellular response. The downstream cellular response. Phosphatase activity, dimerization, and the downsteam cellular response.When nerve cells contact each other, they form adherens junctions (AJs) at the point of contact. The main transmembrane (TM) protein in the junctions is N-cadherin. N-cadherin is a single pass TM protein with a short intracellular domain and a large extracellular domain on the amino end. Proper formation of the AJs requires both actin (MF) and microtubules (MTs). Motor proteins for MTs are dynein (-) end and kinesin (+) end (and were discovered by Michael Sheetz). Maintenance of preformed AJs requires MFs . Adding drugs that disrupt MFs causes AJs to fall apart, and the N-cadherin is removed from the PM (by endocytosis). If the drugs are removed, MTs (& MFs) are required to restore the N-cadherin to the plasma membrane. choose answer for D1 and D2 D. Suppose cells contain a soluble kinase that modifies N-cadherin that has been removed from the PM. D-1. The kinase could catalyze modification(s) of the side chains of amino acids near (the N end) (the C end) (either end, but not…
- Familial hypercholesterolemia (FH) is an inherited disease characterized by high blood levels of cholesterol, xanthomas (lipid-laden nodules that develop under the skin near tendons), and early-onset atherosclerosis (the formation of yellowish plaques within arteries). In the milder form of this disease, patients have half the plasma membrane low-density lipoprotein (LDL) receptors needed for cells to bind to and internalize LDL (a plasma lipoprotein particle that transports cholesterol and other lipids to tissues). These individuals have their first heart attacks in young adulthood. In the severe form of FH, in which affected individuals have no functional LDL receptors, heart attacks begin at about age 8, with death occurring a few years later. Based on what you have learned in this chapter, briefly describe the cellular processes that are defective in FH.GTP binding proteins are molecular switches. How do GTP binding proteins work? Provide two examples of GTP binding proteins that function in intracellular protein transport. Make a drawing that illustrates the function of each of these proteins in their respective roles. Predict the direct outcome of a mutation that: Inhibits GTPase activity Inhibits interaction with the GEFA defective autophagic system in a cell may result in p62 accumulation, inhibition of nuclear factor kappa B, promote chronic inflammation and cause liver tumorigenesis. Group of answer choices True False
- G protein coupled receptors play an important role in signal transduction in many cells. Label the four essential components of the G protein coupled receptor signaling system (blanks a-d in the picture) by choosing from the menus below. a b b Each answer will be used at most once, while some will not be used at all (select one for each): Group of answer choices transcription factor с transcription factor Show Transcribed Text d transcription factor B C. transcription factor G protein second messenger G protein second messenger IE G protein second messenger G protein second messenger receptor receptor receptor receptor enzyme enzyme enzyme enzyme steroid hormone steroid hormone steroid hormone steroid hormoneMetabolic syndrome is a clustering of various conditions including high blood pressure, high blood sugar, excess body fat, high serum triglycerides, and low high-density lipoproteins. Scientists studying metabolic syndrome cultured fibroblast cells from a metabolic syndrome patient and a healthy volunteer and challenged the cells to 100 nM insulin. Ten minutes after the stimulus was applied, they used a protein immunoblot to measure the amount of phosphorylated insulin receptor in the cultures. Use the passage to answer the question. Some patients with metabolic syndrome can continue to gain weight even if they consume fewer calories than other human adults. Based on this finding, which conclusion is MOST likely true? A. Patients with metabolic syndrome do not eliminate as many calories during excretion. B. Patients with metabolic syndrome do not expend as much energy during muscle contractions. C. Patients with metabolic syndrome have more efficient…Metabolic syndrome is a clustering of various conditions including high blood pressure, high blood sugar, excess body fat, high serum triglycerides, and low high-density lipoproteins. Scientists studying metabolic syndrome cultured fibroblast cells from a metabolic syndrome patient and a healthy volunteer and challenged the cells to 100 nM insulin. Ten minutes after the stimulus was applied, they used a protein immunoblot to measure the amount of phosphorylated insulin receptor in the cultures. Use the passage to answer the question. Insulin, in addition to regulating blood glucose, also promotes satiety. Ghrelin, on the other hand, is a peptide hormone that promotes hunger. Which statement BEST predicts the effect of ghrelin release on the insulin receptor? A. It increases insulin receptor expression. B. It decreases insulin receptor expression. C. It increases insulin receptor phosphorylation. D. It decreases insulin receptor phosphorylation.