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Complete the diagrams below to reflect the proteins and any relevant cofactors (e.g. - nducer/repressor molecules) that you would expect to be occupying this operon's regulator region in the presence of low vs high fatty acyl COA levels. [hexose], [fatty acyl CoA] = negligible [hexose] = negligible, ↑[ fatty acyl CoA] promoter operator promoter operator A mutation has occurred rendering the regulatory protein unable to recognize the operator

Complete the diagrams below to reflect the proteins and any relevant cofactors (e.g. - nducer/repressor molecules) that you would expect to be occupying this operon's regulator region in the presence of low vs high fatty acyl COA levels. [hexose], [fatty acyl CoA] = negligible [hexose] = negligible, ↑[ fatty acyl CoA] promoter operator promoter operator A mutation has occurred rendering the regulatory protein unable to recognize the operator

Human Anatomy & Physiology (11th Edition)
Human Anatomy & Physiology (11th Edition)
11th Edition
ISBN:9780134580999
Author:Elaine N. Marieb, Katja N. Hoehn
Publisher:Elaine N. Marieb, Katja N. Hoehn
Chapter1: The Human Body: An Orientation
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Problem 1RQ: The correct sequence of levels forming the structural hierarchy is A. (a) organ, organ system,...
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please help with e f and g

5f. Complete the diagrams below to reflect the proteins and any relevant cofactors (e.g. - inducer/repressor molecules) that you would expect to be occupying this operon's regulatory region in the presence of low vs high fatty acyl CoA levels. ↑ [hexose], [fatty acyl CoA] = negligible [hexose] = negligible, T[ fatty acyl CoA] promoter operator promoter operator 5g. A mutation has occurred rendering the regulatory protein unable to recognize the operator sequence. What effect would this have on the expression of this operon? How would this impact the energetic demands on the cell? Justify your answer.
Transcribed Image Text:5f. Complete the diagrams below to reflect the proteins and any relevant cofactors (e.g. - inducer/repressor molecules) that you would expect to be occupying this operon's regulatory region in the presence of low vs high fatty acyl CoA levels. ↑ [hexose], [fatty acyl CoA] = negligible [hexose] = negligible, T[ fatty acyl CoA] promoter operator promoter operator 5g. A mutation has occurred rendering the regulatory protein unable to recognize the operator sequence. What effect would this have on the expression of this operon? How would this impact the energetic demands on the cell? Justify your answer.
Remember that hexose kinase is an enzyme required for hexose metabolism, a process that takes a 6-carbon sugar and breaks it down into 2 pyruvate molecules. 5a. What specific metabolic process have we studied in which hexose metabolism occurs? 5b. How many steps does this pathway consist of? steps 5c. Overall, this metabolic process (hexose → 2 pyruvate) is exergonic. However, if you look at the individual reactions that makeup this process, they are a mix of endergonic and exergonic reactions. Is the preceding statement true or false? If you selected true, describe the contribution of endergonic reactions to the overall exergonic process. If false, explain why. You discover that rather than being hormonally regulated (as seen above in humans), hexose kinase is part of an operon in prokaryotic organisms. This operon is known as the "hexose operon" and it regulates expression of the complete set of enzymes needed to convert a hexose sugar into 2 pyruvate molecules. 5d. Remembering that hexose metabolism is a highly conserved process among all branches of life, how many genes do you expect to find in this operon? Explain your logic. Like the lac operon, the hexose operon is controlled by a separate regulatory protein under the control of its own promoter (see the schematic of the operon below). The hexose regulatory protein is sensitive to fatty acyl CoA levels. When all hexose fuel sources are depleted, the bacteria switch to lipid metabolism and fatty acyl CoA levels increase. This turns expression of the hexose operon off. +1 +1 Regulatory Gene Pregulator Poperon Hexose Operon Genes regulator promoter operon promoter 5e. The regulatory protein that controls expression of the hexose operons is a transcriptional ACTIVTOR or REPRESSOR (circle one).
Transcribed Image Text:Remember that hexose kinase is an enzyme required for hexose metabolism, a process that takes a 6-carbon sugar and breaks it down into 2 pyruvate molecules. 5a. What specific metabolic process have we studied in which hexose metabolism occurs? 5b. How many steps does this pathway consist of? steps 5c. Overall, this metabolic process (hexose → 2 pyruvate) is exergonic. However, if you look at the individual reactions that makeup this process, they are a mix of endergonic and exergonic reactions. Is the preceding statement true or false? If you selected true, describe the contribution of endergonic reactions to the overall exergonic process. If false, explain why. You discover that rather than being hormonally regulated (as seen above in humans), hexose kinase is part of an operon in prokaryotic organisms. This operon is known as the "hexose operon" and it regulates expression of the complete set of enzymes needed to convert a hexose sugar into 2 pyruvate molecules. 5d. Remembering that hexose metabolism is a highly conserved process among all branches of life, how many genes do you expect to find in this operon? Explain your logic. Like the lac operon, the hexose operon is controlled by a separate regulatory protein under the control of its own promoter (see the schematic of the operon below). The hexose regulatory protein is sensitive to fatty acyl CoA levels. When all hexose fuel sources are depleted, the bacteria switch to lipid metabolism and fatty acyl CoA levels increase. This turns expression of the hexose operon off. +1 +1 Regulatory Gene Pregulator Poperon Hexose Operon Genes regulator promoter operon promoter 5e. The regulatory protein that controls expression of the hexose operons is a transcriptional ACTIVTOR or REPRESSOR (circle one).
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