The pancreas is an organ of mixed secretion. Endocrinely, beta-cells produce the hormone insulin, which affects carbohydrate metabolism. How it affects the blood glucose level. Describe the mechanism of action
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The pancreas is an organ of mixed secretion. Endocrinely, beta-cells produce the hormone insulin, which affects carbohydrate
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- Figure 37.11 Pancreatic tumors may cause excess secretion of glucagon. Type I diabetes results from the failure of the pancreas to produce insulin. Which of the following statement about these two conditions is true? A pancreatic tumor and type I diabetes will have the opposite effects on blood sugar levels. A pancreatic tumor and type I diabetes will both cause hyperglycemia. A pancreatic tumor and type I diabetes will both cause hypoglycemia. Both pancreatic tumors and type I diabetes result in the inability of cells to take up glucose.The pancreas has both endocrine and exocrine functions as evidenced by: O Alpha and beta cells that secrete their hormones into a capillary network and acinar cells that secrete enzymes into the small intestine. O Alpha, beta and acinar cells that all secrete both enzymes and hormones into the pancreatic duct and bloodstream. O Alpha and beta cells that secrete their hormones into the small intestine and acinar cells secrete hormones into the small intestine. O. Acinar cells that secrete their hormones into a capillary network and alpha and beta cells that secrete enzymes into the small intestine.Which part of the pancreas has the highest blood pressure. A tpu scaffold will be placed around the pancreas for diabetic patients to infuse islet beta cells held within the scaffold into the pancreas. Where would blood pressure affect the scaffold the most causing deformation of the scaffold. Also what other factors will affect the scaffold's life cycle?
- Which of the followings is an example of continuous secretion? Release of intestinal mucous Release of zymogen from pancreas O Release of insulin hormone Release of neurotransmitter in synapseDescribe the negative feedback control of blood glucose levels, and explain the roles of insulin and glucagon in regulating blood glucose levels. ANSWER SHOULD INCLUDE: Explain the meaning of "negative feedback control', Name the 2 hormones involved and which part of pancreas produces each one Explain the stimulus for release of each hormone. Explain the response to hyperglycaemia (what happens to glucose? Explain the meaning of term such as facilitated diffusion, glycogenesis , gluconeogenesis) Explain how blood glucose returns to normal (homeostasis) Explain the response to hyperglycaemia (what happens to glucose? What happens in glyconeogenesis?) In which organs do these events occur? Explain how blood glucose level returns to normal (homeostasis)Pancreatic acinar cells produce and store hydrolytic enzymes in zymogen granules until hormones signal the cell to release the enzymes in a process known as 00 transport secretion. polarized secretion. regulated secretion. constitutive secretion.
- The control of pancreatic, liver, and intestinal secretion is achieved in part by hormones secreted by the: Group of answer choices gall bladder pancreas esophagus small intestines stomachSecretory cells of the mucous membrane of the mucosal layer of the gastrointestinal tract (GI tract) are classified as ___ cells if they secrete fluids and enzymes into the lumen and ___ cells if they release hormones into the bloodstream. Group of answer choices exocrine : endocrine endocrine : absorptive endocrine : exocrine exocrine : absorptiveWhich weight loss suggestion is designed to achieve some level of ketosis? Atkins Diet Exercise High fiber diet Herbal treatments
- The pancreas produces tiny amounts of insulin 24 hours a day. This small amount of insulin keeps glucose levels stable between meals and during sleep. This tiny continuous production of insulin is called:a) Basal insulinb) Bolus insulinAbnormal triglyceride accumulation in the liver (hepatic steatosis) leads to insulin resistance, which is trademark trigger of type 2 diabetes. Insulin-resistant hepatocytes perform gluconeogenesis even in the presence of high blood glucose levels, which exacerbates the hyperglycemia. These insulin-resistant cells also perform excessive b-oxidation, which can lead to ketoacidosis. Insulin normally suppresses lipolysis, and thus insulin-resistant adipocytes release too many fatty acids into circulation, many of which are taken up by hepatocytes and esterified into triglyceride. Thus, insulin resistance further exacerbates hepatic steatosis, which will in turn worsen insulin resistance in the liver. Given this vicious cycle, abatement of hepatic steatosis is one of the mainstays of diabetes treatment. Metformin is one such treatment and works by activating fatty acid degradation and inhibiting fatty acid synthesis. Which of the following could be the mechanism of action of metformin? (A)…Visit this link (http://openstaxcollege.org/l/pancreas1)to view an animation describing the location and functionof the pancreas. What goes wrong in the function of insulinin type 2 diabetes?
