4. A 39-year-old patient was presented with persistent arterial hypertension(200/120 mm Hg), which was not eliminated by conventional antihypertensivedrugs. An ultrasound of the renal arteries revealed signs of stenosis of the rightrenal artery. Why does renal artery stenosis lead to an increase in blood pressure.For explanation:a) present a chart of water and salt balance regulation, indicate the main stimuliof system activation and the final effects;b) name the compound, with its increased synthesis caused by a drop in renalarteries perfusion pressure;c) describe the consequences arising from an increase in the blood concentrationof this substance, using the scheme presented in paragraph a).

Renal artery stenosis is more likely if:• Hypertension is severe, or of recent onset, or difficult…

Answered: a) present a chart of water and salt…

Phlebotomy Essentials

6th Edition

ISBN:9781451194524

Author:Ruth McCall, Cathee M. Tankersley MT(ASCP)

Publisher:Ruth McCall, Cathee M. Tankersley MT(ASCP)

Chapter1: Phlebotomy: Past And Present And The Healthcare Setting

Section: Chapter Questions

Problem 1SRQ

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Similar questions

3. In a normal frequency distribution of serum sodium concentrations, the mean is 141 mEq/L and the standard deviation is 3.2 mEq/L. Which of the following most closely represents the percentage of values in the interval between 137.8 mEq/L and 144.2 mEq/L? A) 25% B) 50% C) 65% D) 95% E) 99%
S3. Diuretics can be prescribed to combat high blood pressure. Thiazide is one of the most common. Describeits mechanism of action (they physiology of what the drug does) and how it combats high blood pressure.?
Describe the mechanism of action of diuretics most commonly used for hypertension treatment. What are the major side effects and drug-drug interactions?
1. Name the other methods of ESR determination with its corresponding anticoagulants and normal values. 2. What are the factors favoring slow ESR? NOTE: Kindly answer all question. Thank you!
During treatment of congestive heart failure and hypertension, describe the mechanism of action and major side effects of loop diuretics, ACE inhibitors, blockers, and Ca2+ channel blockers.
1) Ordered ; Ceftazidime 19 IM qbh to be administered In 50ML bag of Ns over 30 minutes. Available : ceftazidime 2gram Cpowder) Reconstitute drug with lomL of diluento o)ose strength after recorstitution is ( 200 mg/ml). Administer dose per N flow rate
8. A 45-year-old man comes to the physician for a routine health maintenance examination. His blood pressure is 150/90 mm Hg. Physical examination shows no other abnormalities The physician advises the patient to decrease sodium intake. Two weeks after beginning a low-sodium diet, which of the following is most likely to be decreased in this patient? OA) Plasma renin activity B) Serum aldosterone concentration C) Serum angiotensin II concentration D) Serum potassium concentration OE) Serum sodium concentration
4) Explain why the edema of tissues does not appear if it was stated that the filtration force which ensures the exit of liquid from the arterial end of the capillary, normally exceeds the force of reabsorption, which ensures the entry of liquid into the venous end, by 1 mm Hg.
3) A solution containing 500,000 units of polymyxin B sulfate in 20 mL of sterile water for injection is added to 500 mL of 5% dextrose iniection The infusion is to be administered over 2.5 hours. If the administration set calibrates 10 drops/mL. At what rate, in drops per mindte, should the flow be adjusted to administer the infusion over the designated time interval? (Do not ignore volume contribution due to admixture). ean
3.. The suggested dose of drug X is 75mg by slow infusion at a rate not surpassing 0.3 g/hour. Drug C is available as an injection containing 15mg of drug X in 1 mL. Calculate the infusion rate in mL/minute.
6) "The health care provider prescribed 1.5 L of 5% dextrose in 0.9% normal saline to infuse over 24 hours for a newly-admitted client...". Drop factor is 20 gtt/mL. How many drops per minute? Answer:
Describe the therapeutic strategies that target angiotensin II to treat Stage 1 and 2 hypertension. With the aid of diagrams describe the aetiology of the disease. Pick one therapeutic approach and describe in detail the mechanism of action and major side effects.

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